Journal Of Experimental Neurology

Journal Of Experimental Neurology – UF Associate Professor Eduardo Candelario-Jalil, Ph. D. and his team identified emerging neuroprotective strategies for the treatment of ischemic stroke in a summary of clinical and preclinical studies. This study provides an update on recent advances in the development of new stroke treatments.

This invited review article is part of a special issue of Experimental Physiology focusing on disorders of ischemic stroke. “We summarize recent advances and failures in clinical trials in ischemic stroke, and discuss ongoing efforts to develop new treatments for ischemic stroke,” explained Candelario-Jalil.

Journal Of Experimental Neurology

Journal Of Experimental Neurology

Candelario-Jalil and his team are researching treatments for a common type of stroke known as ischemic stroke, caused by a sudden blockage of a blood vessel. A less common type of stroke is caused by bleeding in the brain when a blood vessel ruptures. This is known as a blood clot.

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Heart failure is common in all countries. It is the leading cause of disability and the second leading cause of death worldwide. “With the global population aged 65 and over growing faster than any other age group, the number of attacks is also increasing.” Research also suggests that there is a change in the burden of assault in youth groups, especially in low- and middle-income countries. This can be due to several reasons, including a lack of knowledge in society about the risk of stroke, warning signs, and other important information related to the brain.

Although treatment options for stroke are limited, there is hope with advances in stroke care, where some progress has been made in helping patients recover from ischemic stroke. Despite this, “there is still a great need for the development of therapeutic agents for neuroprotection in acute ischemic stroke to protect the brain from damage before and during rehabilitation, to expand the therapeutic window for intervention, and will further improve outcomes action.”

“We have studied in detail the neural mechanisms involved in the death of the neural tissue after ischemic stroke, as well as the molecular mechanisms underlying neuroprotection with different treatments, which can be help in the development of future therapeutic strategies is ‘lu,'” Candelario-Jalil concluded. Share to Twitter Share to Facebook Share to Reddit Share to Tumblr Share to Pinterest Share to Email

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